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303 E. Superior St.

Lurie 7-125

Chicago, IL 60611

 

676 N. Saint Clair St.

Suite 1260

Chicago, IL 60611

 

303 E. Chicago Ave.

Ward 9-148

Chicago, IL 60611

 

Ph: 312.503.5600

Fax: 312.503.5603

 

Faculty

 

Erik J. Sontheimer, PhD
Assistant Professor
Department of Biochemistry, Molecular Biology and Cell Biology

To Contact Dr. Sontheimer:
phone: 847-467-6880
e-mail: erik@northwestern.edu
Dr. Sontheimer's website
PubMed Reference Lookup


Research Interests
The laboratory of Dr. Erik Sontheimer studies the functions of RNA molecules in eukaryotic gene expression. Pre-mRNA splicing (the accurate removal of introns from premessenger RNA) is mediated by several small nuclear RNAs, and is essential for the expression of most eukaryotic genes. Many inherited diseases result from defects in the removal of introns from specific transcripts; approximately 15-20% of point mutations that cause human disease do so by disrupting splicing. Several mechanistic aspects of pre-mRNA splicing are poorly understood. How is the fidelity of splice site selection maintained? How do specific protein and RNA factors collaborate to mediate splicing complex assembly? What are the components of the splicing machinery’s catalytic core” that catalyze the chemical reactions of pre-mRNA splicing? Dr. Sontheimer’s lab uses biochemical approaches to addresss these issues and thereby obtain a deeper understanding of intron removal. In addition to their studies of pre-mRNA splicing, they are investigating the function of the 7SK small nuclear RNA in transcriptional elongation. The transcription of numerous loci such as the myc oncogene, the heat shock genes, and the Human Immunodeficiency Virus (HIV) genome are known to be controlled at the level of elongation rather than (or in addition to) initiation. The 7SK RNA was recently shown to inhibit the activity of the transcription elongation factor P-TEFb, but the mechanism of 7SK’s function is unknown. They are using Xenopus oocytes as an experimental system to investigate the 7SK RNA and its role in controlling transcriptional elongation by RNA polymerase II.

 


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