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303 E. Superior St.

Lurie 7-125

Chicago, IL 60611

 

676 N. Saint Clair St.

Suite 1260

Chicago, IL 60611

 

303 E. Chicago Ave.

Ward 9-148

Chicago, IL 60611

 

Ph: 312.503.5600

Fax: 312.503.5603

 

Faculty


Robert Lavker, PhD

Professor of Dermatology
Director, Dermatology Research


To contact Dr. Lavker:
Phone: 312-503-4315
e-mail: r-lavker@northwestern.edu
Dr. Lavker's website

PubMed Reference Lookup

Research Interests
Dr. Lavker's laboratory has centered on investigations of processes of proliferation and differentiation in keratinizing tissues. His laboratory in conjunction with Tung-Tien Sun's laboratory at NYU was the first to demonstrate that stem cells of the corneal epithelium were located within the limbal epithelium. This finding has had substantial impact on corneal epithelial transplantation for treating patients with damage to the ocular surface. The fornical epithelium has also been demonstrated to be enriched in conjunctival epithelial stem cells. With respect to the skin, Drs. Lavker and Sun were the first to demonstrate the existence of stem cells at the tips of the deep rete ridges of palm epidermis, and in the bulge region of the outer root sheath epithelium of the hair follicle. The latter observation formed the basis for a new hypothesis on the mechanism(s) involved in the regulation of the hair growth cycle. More recently, novel techniques have been developed that can selectively tag subpopulations of proliferating cells, and the hair follicle has been shown to be a major repository of skin keratinocyte stem cells. This indicates that the bulge follicular stem cells are bipotent because they give rise not only to the hair follicle but also to the epidermis.

The Lavker laboratory also has a long-standing interest in studying the intrinsic and extrinsic effects of age on the structure of the epidermis and dermis. To this end, the laboratory has described chronic inflammatory changes in photodamaged skin and provided evidence of mast cell-fibroblast interactions. More recently the laboratory has focused on the role of ultraviolet A (UVA) in the etiology of photodamaged skin. Findings suggest that UVA may contribute significantly to long-term actinic damage and underscore the need for effective broad UVA-blocking sunscreens.



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